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THE MERCK MANUAL MEDICAL LIBRARY: The Merck Manual of Diagnosis and Therapy
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Hernias of the Abdominal Wall

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A hernia of the abdominal wall is a protrusion of the abdominal contents through an acquired or congenital area of weakness or defect in the wall. Many hernias are asymptomatic, but some become incarcerated or strangulated, causing pain and requiring immediate operation. Diagnosis is clinical. Treatment is elective surgical repair.

Abdominal hernias are extremely common, particularly in males, necessitating about 700,000 operations each year in the US.

Classification

Abdominal hernias are classified as either abdominal wall or groin hernias. Strangulated hernias are ischemic from physical constriction of their blood supply. Gangrene, perforation, and peritonitis may develop. Incarcerated and strangulated hernias cannot be reduced manually.

Abdominal wall hernias include umbilical hernias, epigastric hernias, Spigelian hernias, and incisional (ventral) hernias. Umbilical hernias (protrusions through the umbilical ring) are mostly congenital, but some are acquired in adulthood secondary to obesity, ascites, pregnancy, or chronic peritoneal dialysis. Epigastric hernias occur through the linea alba. Spigelian hernias occur through defects in the transversus abdominis muscle lateral to the rectus sheath, usually below the level of the umbilicus. Incisional hernias occur through an incision from previous abdominal surgery.

Groin hernias include inguinal hernias and femoral hernias. Inguinal hernias occur above the inguinal ligament. Indirect inguinal hernias traverse the internal inguinal ring into the inguinal canal, and direct inguinal hernias extend directly forward and do not pass through the inguinal canal. Femoral hernias occur below the inguinal ligament and go into the femoral canal.

About 75% of all abdominal hernias are inguinal. Incisional hernias comprise another 10 to 15%. Femoral and unusual hernias account for the remaining 10 to 15%.

Symptoms and Signs

Most patients complain only of a visible bulge, which may cause vague discomfort or be asymptomatic. Most hernias, even large ones, can be manually reduced with persistent gentle pressure; placing the patient in the Trendelenburg position may help. An incarcerated hernia cannot be reduced but has no additional symptoms. A strangulated hernia causes steady, gradually increasing pain, typically with nausea and vomiting. The hernia itself is tender, and the overlying skin may be erythematous; peritonitis may develop depending on location, with diffuse tenderness, guarding, and rebound.

Diagnosis

  • Clinical evaluation

The diagnosis is clinical. Because the hernia may be apparent only when abdominal pressure is increased, the patient should be examined in a standing position. If no hernia is palpable, the patient should cough or perform a Valsalva maneuver as the examiner palpates the abdominal wall. Examination focuses on the umbilicus, the inguinal area (with a finger in the inguinal canal in males), the femoral triangle, and any incisions that are present.

Inguinal masses that resemble hernias may be the result of adenopathy (infectious or malignant), an ectopic testis, or lipoma. These masses are solid and are not reducible. A scrotal mass may be a varicocele, hydrocele, or testicular tumor. Ultrasound may be done if physical examination is equivocal.

Prognosis

Congenital umbilical hernias rarely strangulate and are not treated; most resolve spontaneously within several years. Very large defects may be repaired electively after age 2 yr. Umbilical hernias in adults cause cosmetic concerns and can be electively repaired; strangulation and incarceration are unusual but, if happen, usually contain omentum rather than intestine.

Treatment

  • Surgical repair

Groin hernias should be repaired electively because of the risk of strangulation, which results in higher morbidity (and possible mortality in elderly patients). Repair may be through a standard incision or laparoscopically.

An incarcerated or strangulated hernia of any kind requires urgent surgical repair.

Last full review/revision September 2007 by Parswa Ansari, MD

Content last modified September 2007

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